The data shows that meprin metalloproteases are involved in general tissue differentiation.
A Morpholino knockdown in zebrafish embryos targeting meprin α1 and β mRNA caused defects in general tissue differentiation.
In general, tissue sections stained by hematoxylin and eosin (H&E; see below) were examined by light microscopy (BX51, Olympus, Japan).
In general, tissue sampling was the preferred reference standard.
In general tissue debris does not linger for long periods after tissue damage due to efficient removal by macrophages.
In general, tissue pieces were stored in washing buffer (PBS with 1-2% (v/v) Pen/Strep and 1-21-2%/v) AmPAAterinin B (PAA)) in the refrigerator or on ice until isolation.
Some subtilase genes, such as LOC100260464, LOC100243364, LOC100248833, LOC100243797 and LOC100265217, are present at a ubiquitously high level in roots, leaves, stems, floral buds and internodes, indicating the role of subtilases in general tissue growth and development.
These networks were, in general, tissue-specific.
In general, tissues in thin-sample transmission geometry were seen to be more depolarizing than phantoms for both linear and circular polarization states, an effect tentatively attributed to dependent scattering mechanisms [ 21– 21].
In general, tissues with a high rate of cell division may display more age-related epigenetic variation through stochastic errors in maintaining and transmitting epigenetic information than tissues with lower rates of cell division (Thompson et al., 2010).
