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Both hypotheses, together with other key theories of climate-evolution linkages, are discussed in this paper.
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Table 2 sums up our analyzed proxy variables for each of the hypotheses together with the hypothetical signs and variable definitions.
The phylogeny, dispersal tracts and time markers associated with this hypotheses, together with additional references and details are further explained by Table 1 and Figures 2B and 3B.
Such hypotheses of course need to be verified epidemiologically through cohort studies of incidence and/or through prevention trials that test these hypotheses together with testing ways to apply them in practice.
Our hypotheses, together with general assumptions about tuning curve shape and decoding mechanism, explain the triangular lattice structure of two-dimensional grid cell firing maps and predict a geometric progression of grid scales.
The rationale and reasoning for how the data support (or do not support) each account's hypotheses, together with the plausibility of subsidiary explanations or assumptions in view of wider biological knowledge, constitute the basis for evaluating the scientific support each account gets from available data.
The first hypothesis, together with its sub-hypotheses, is based on the above factors.
This yields y (ν+1)≤y, again exploiting the induction hypothesis together with the fact that f is monotonically decreasing.
For, likelihoods represent the empirical content of a hypothesis, what the hypothesis (together with background b) probabilistically implies about the evidence.
This hypothesis, together with surface precipitation of Al–(P HCA chelates, is also useful to interpret GLU and TAR surface concentrations at PHCA equilibrium concentrations ≥ 0.1 mol L−1.
However, support-function likelihoods are supposed to represent what the hypothesis (together with explicit background and experimental conditions) says or implies about the evidence.
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