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Regardless of follow-up length, the non-DM subjects remaining in the study lost a significant amount of weight in both groups with significantly greater weight loss in the PMR-treated subjects.
Kinetics of CI, SVI, MAP, HR, and GEVI showed similar patterns in both groups with significantly higher values in the HES group at the end of the experiment.
Kinetics of the CI, MAP, HR, and GEDI showed similar pattern in both groups with significantly higher values in the HES group at the end of the experiment (T 4).
CD62L-positive PMNs decreased while CD62L-positive monocytes increased in both groups with significantly lower values in the verum-group.
RSV-related hospitalizations prior to the index palivizumab dose occurred in both groups, with significantly more hospitalizations occurring before the index dose in the non-compliant group.
Self-measured plasma glucose levels decreased in both groups, with significantly greater reductions in postprandial values in the insulin detemir versus control group (Fig. 2 D ).
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Body weight increased in both groups, with a significantly greater increase in the rosi + met group [mean difference 1.1 (0.2.12.1) kg (P = 0.021) Table 2].
Psychotic features and agitation and aggression, assessed by PANSS total and positive and activation subscales, were improved in both treatment groups, with significantly greater improvements in the add-on lithium than add-on placebo group (p < 0.001, 0.003, and < 0.001, for respective scales; Table 2).
IL-17A mRNA levels correlated with disease activity in each individual in both diet groups with significantly reduced levels close to those of controls after GFD.
Both groups with limiting LTCs were significantly more likely to report worse experiences after accounting for these other factors: S-LLTC group (OR = 1.23; 95% CI: 1.03 to 1.48) and M-LLTC group (OR = 1.64; 95% CI: 1.19 to 2.26).
The α-TL conjugates in both groups with diabetes remained highly significantly increased compared to the control group, whereas, with the exception of α-CEHC sulfate in the well controlled diabetic group, the concentrations of the α-CEHC conjugates were no longer significantly different from the controls.
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