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In both groups, the renal function did not significantly change during the experimental period.
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In both groups, the strongest determinants of renal decline were baseline serum concentrations of uric acid (P < 0.001) and tumor necrosis factor receptor 1 or 2 (TNFR-1 or -2, P < 0.001).
There was no difference in the degree of rise in renovascular resistance between both groups, indicating that the renal nerves do not play a role in altering renal vascular resistance with the application of CPAP.
There was no difference in the extent of the increase in renovascular resistance in response to CPAP between both groups suggesting that the renal nerves do not play a role in altering renal vascular resistance with the application of CPAP.
In both groups, renal function parameters showed an increase in creatinine and urea during treatment with L-AmB, but not significantly beyond the upper normal limit.
In all groups but the renal controls, more than 85% of the individuals were women.
As compared with the Control group, the renal-to-systemic vascular resistances index more than doubled in the AKI groups.
According to Griffin classification of renal failure, mild to moderate degree of renal failure was observed in both of the renal failure groups as indicated by results of tubular and glomerular functions (Table 4) and histology study.
Survivors had a similar renal recovery in both groups, but the hospital length of stay was longer in the delayed group (51 vs 34 days, P = 0.05).
Malondialdehyde, an index of lipid peroxidation, increased while total glutathione was decreased in the liver in both the renal ischemia and nephrectomy groups, suggesting activation of oxidative stress.
The mean standardized height (SD score) was –4.0 in the conservative treatment group, –4.4 in the dialysis group, and –4.9 in the renal transplant group.
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CEO of Professional Science Editing for Scientists @ prosciediting.com