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During dark trials, both groups of rats dug equally quickly in both the go and no-go trials (Sham1 mean latency to dig go trials = 6.18 s, no-go trials = 6.51 s; RScomb mean latency to dig go trials = 4.83 s, no-go trials = 4.94 s).
Both groups of rats (morphine- and placebo-pelleted) showed a CPP after initial conditioning with morphine.
Following stress, the sleep architecture of both groups of rats remained unaltered in block 1 relative to their baseline day.
K+ induced a similar release of noradrenaline from the hypothalamus in both groups of rats (239% in sham animals and 283% in the subarachnoid haemorrhage group).
Two and a half hours after the induction of inflammation, in both groups of rats with unilateral lesion, paw withdrawal latencies decreased significantly in the LC/SC-lesioned rats.
Pharmacological challenges with the NMDA antagonists, PCP and ketamine, and with the dopamine modulator, amphetamine, decreased accuracy to a similar extent in both groups of rats when intertrial delays were held constant at 10 s; however, nicotine did not decrease accuracy in either group.
Similar(20)
Although 2 weeks of ABS was insufficient to restore body weight, both groups of abstinent rats had increased plantaris CSAs compared with muscles from alcohol-fed rats (P ≤ 0.05).
Both groups of aged rats exhibited delayed ABR latencies (III, IV, V), MLR Pa latency, and I-IV interpeak latency.
Both groups of lesioned rats presented a deficit in the non-visible platform task but not in the visible platform one.
In addition, SUP-males and SUP-females exhibited a reduction in burst responding (response latencies <2 sec) compared with both groups of CON rats.
The number of pericytes was found significantly lower in both groups of diabetic rats than in control animals.
More suggestions(15)
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com