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Consistent with the results shown in Figure 1A, the VEH and ANI groups displayed enhancement and disruption, respectively, of the reactivated memory at PR-LTM, although both groups displayed comparable crossover latency at Reactivation compared to the VEH and ANI groups, respectively, in Figure 1A.
Seven days after reperfusion, both groups displayed comparable residual levels of damages (figure 2).
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The ANI groups displayed comparable crossover latency at PR-LTM-1 compared with Reactivation.
All groups displayed comparable haemostasis at 24 h post-surgery.
The increases were surprisingly short-lived; all groups displayed comparable plasma fibrinogen and FIBTEM MCF by 24 h post-surgery and during the subsequent acute-phase response.
Interestingly, protein synthesis inhibition in the hippocampus or mPFC failed to disrupt the reactivated IA memory, but blocked its enhancement; the ANI groups displayed comparable crossover latency between Reactivation and PR-LTM.
Moreover, given the clinicopathological differences between the cohorts, the PPP2R2A (B55α) scoring methodology was able to define groups displaying comparable magnitudes of HR, thus further reinforcing the reproducibility of the method.
Though both groups displayed high and comparable accuracy rates upon performing the rather easy experimental tasks during scanning, dyscalculic children were slower than controls upon making number magnitude classifications, which is in line with the literature [ 3, 4].
Both groups displayed a relatively low 'other-esteem'.
Both groups displayed a similar total range of knee motion.
Both groups displayed MGMT promoter hypermethylation.
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