Curiously, both of these defects can be rescued by overexpression of Ndel1, showing a redundancy and/or mutual dependency between Lis1 and Ndel1 (Yingling et al., 2008; Moon et al., 2014).
Both surface and subsurface defects can be detected at a high efficiency.
The strength of the proposed approach is that both structural and textural surface defects can be visually enhanced, detected, and well separated from acceptable surfaces.
Defects can be engineered (both deliberately and serendipitously) in MOFs to produce materials with improved function for adsorption, catalysis, etc.
Gaussian analysis of the emission data indicates that subband states change with anodizing parameters, and various point defects can be formed both in the bulk and on the surface of these nanoporous layers during anodizing.
Such defects can be electrically charged, both in the bulk and on the surface.
Heterozygous persons do not express the phenotype; therefore, NIS gene defects can be detected only when both alleles are affected.
This is supported by the fact that both the lin-17/Fz and bar-1/β-catenin mutant defects can be rescued cell-autonomously in D-type neurons.
These defects can be specific to TC-NER, GG-NER or both sub-pathways of NER [ 14, 15].
Septal defects can be repaired surgically.
