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Generally, members of both clusters have been implicated in malignant growth.
miRNAs belonging to both clusters have been considered to act as either a tumour suppressor or an oncogene depending on the tumour type (O'Donnell et al, 2005; Garofalo et al, 2012).
As observed with the characterized cluster, the second cluster (A1S_0938-0940) is highly conserved across most strains; both clusters have been removed by insertion-deletion events in strain SDF (Table 3).
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Both miRNA clusters have been shown to accumulate in different types of cancer and have been designated oncomiRs, with miR-17-92 miR-17-92 miR-17-92comiR-1 (He et al, 2005).
As gene expression in clusters 1 to 5 displayed similar expression profiles in the control female group, these clusters have been analysed both all together and separately (Table 1).
These energies for both pure and impure clusters have been reported in Tables 2 and3.
Since both single CTCs and CTC clusters have been detected in venous blood samples of cancer patients, they needed to traverse at least one capillary bed when crossing from arterial to venous circulation.
Details regarding the synthesis and characterization of both molecular wires and nanoscale metallic clusters have been discussed from a perspective that emphasizes the self-assembly of these nearly identical "building blocks" into interesting molecular nanostructures.
Both Gypsy - and Copia-related clusters have been further characterized identifying the LTR-RT families to which they putatively belong.
Neutral and ionic alkali-metal halide clusters have been widely studied, both experimentally and theoretically.
We see that fully half of these clusters have been created through one or both abstractions.
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