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In this work, the effect of the RGD-SBM concentration on both cells migration and proliferation was not evaluated.
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Both cell migration and transfection have been monitored by fluorescence microscopy.
Stable expression of Tensin3 did not affect cell viability and proliferation; however, it affected both cell migration and invasion negatively.
Stable expression of Tensin3 in HEK 293 cells markedly inhibited both cell migration and matrix invasion, a function independent of putative phosphatase activity in Tensin3.
Through our experiments, we confirmed that hUCBSC effect on glioma cells decreased the levels of phosphorylated Akt, which alters both cell migration and wound healing capacity.
Altered expression was found in genes involved in both cell migration and metabolism; modifications in ubiquitin-protein ligase activity are due to alterations in protein binding.
CTGF is, as discussed earlier in the text, involved in both cell migration and EMT.
However, the VEGFR2 has important roles in ELCs for both cell migration and endothelial differentiation.
SB203580, a p38 MAPK specific inhibitor blocked both cell migration and actin polymerization without affecting cell proliferation.
This was confirmed in vitro, as p21 gene silencing in SCP2 cells inhibited both cell migration and invasion.
By disrupting the cytoskeleton through Rho-GTPase modulation, both cell migration and tissue integrity can be influenced.
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