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HRV16 infection in IL-13-treated cells of both cell types did not further increase eotaxin 3 production.
Both cell types did not give rise to tumors in immunocompetent allogeneic mice or xenogeneic rats.
Importantly, the drugs that did attenuate both cell types did so with similar IC50 values suggesting that they are targeting the same cellular factor.
Our finding that trends in survival over time were similar for both cell types did not confirm the earlier observation (Janssen-Heijnen et al, 1998) of a worsening trend with adenocarcinoma.
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Both cell types do not have close contact but are separated by a wide interstitium.
Functional characterization of BM-MSCs and ASCs has shown that both cell types do not express HLA-DR (of the major histocompatibility complex class II, MHC II), which renders these cells significantly less immunogenic than other cell types, while they are also capable of suppressing lymphocyte reactivity in mixed lymphocyte reaction (MLR) assays and reducing inflammation in vivo [ 8, 15, 20– 23].
Although the cell viability studies did not reveal a statistical difference between Ad/ FasL-GFPDiSTRES and control, both prostate cancer cell types did demonstrate the typical cell-rounding morphology as a result of Ad/ FasL-GFPDiSTRES infection (Fig. 6).
Proviral load and reduced glutathione (GSH) levels in leukocyte cell types did not change significantly following supplementation.
Differential localization patterns of Zn II Pz derivatives between cell types did not appear to influence the overall PDT effect.
Omission of a few cell types did not grossly alter the results for the other cell types.
Other cell types did not cleave the biosensor efficiently suggesting that cellular proteinases distinct from furin-like PCs did not contribute significantly to the biosensor cleavage.
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