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Both cell lines display similar proliferation rates [ 20].
Both cell lines display autocrine FGFR signaling activity due to co-expression of ligands and receptors [ 9].
These data suggest that while both cell lines display similar sensitivity to the growth inhibitory effects of HDACi, the HT29 cells are significantly more resistant to the onset of HDACi-induced apoptosis in this time-frame.
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Both cell lines displayed almost 100% infection with Wolbachia but they differed dramatically in the density of bacteria per cell.
Both cell lines displayed comparable levels of Ras modification, indicating that similar amounts of ExoS were translocated into the different cells (Figure 4A).
As shown in Fig. 6C, ribosomes of both cell lines displayed the ability to pass through the premature stop codon although this dysfunctional property was 30% higher in the most aggressive cells MA- cells than the control cells MP.
Both cell lines displayed modest sensitivity to TNF.
However, both cell lines displayed decreased expression of E-cadherin.
Growth kinetics of spheroids from both cell lines displayed an exponential growth phase.
Moreover, both cell lines displayed homozygosity for all tested SNPs (Supplementary information, Figure S3B and Table S3).
Both cell lines displayed an epithelial morphology when cultured in F9-CM or N-CM and E-cadherin and β-catenin localized to the cell membrane.
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