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The multiple regression equations were developed for multiple bones to increase its accuracy.
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Usually, HA is mixed with autologous bone to increase the graft volume with an osteoconductive material.
Therefore, sequential treatment with bFGF and Ris restores trabecular architecture and allows mineralization of bone to increase, which appears to be beneficial to bone strength.
Here we show that 2ME2 also has direct effects on bone to increase bone mass by decreasing osteoclasts and increasing osteoblasts.
PTH acts in bone to increase the number and activity of osteoblasts and osteoclasts, and increases bone turnover.
SOST may need to be further stabilized or targeted to the bone to increase its potency as a treatment.
Released by the chief cells of the parathyroid gland in response to deviations in serum calcium levels, PTH primarily acts on kidney and bone to increase calcium reabsorption and liberate calcium from bone matrix, respectively.
Although these changes likely represent appropriate homeostatic responses of bone to increased loading, they also appear to inadvertently predate maladaptive responses in other articular structures, most notably cartilage.
Increasing levels of BV/TV, modulus and strength with decreasing levels of growth factors in our study may be associated with an adaptive response of bone to increasing mechanical demands in these bone regions.
Site #24 was augmented with guided bone regeneration to increase bone width for implant placement.
In women, the decrease in estrogen due to menopause causes bone resorption to increase by 90% while bone formation increases by only 45%, leading to overall bone loss.
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