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Vascular channels penetrating cortical bone were identified in knees of non-arthritic mice adjacent to the cruciate ligaments.
In addition, the remains of ectopic cartilaginous elements in trabecular bone were identified in ptpn11 fl/fl ert2-cre mice (Fig. 5, top left, middle left and bottom left panels).
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Macroscopically, sickle bone is identified in clinical radiographs by its reduced mineral density, widening of the marrow cavity, and thinning of the cortical bone due to the elevated erythroid hyperplasia accompanying the disease.
Severe suture restriction of the temporal bone was identified in 23 children (35.9%).
Metastatic lesions in bone were identified on scanned images as fluorescent regions.
A significant number of the top-ranking differentially expressed genes in OA bone were identified as WNT signalling pathway targets (Table 3).
Presence or absence of bone bridges Bone bridges were identified in all the treated specimens (Fig. 6e).
Then, hot foci of bone metastases were identified in demarcated sclerotic bone metastatic areas, or in areas that could not be explained by the presence of degenerative changes.
Spot welds consistent with bone ingrowth were identified in all of the femoral components.
Zirconium particles from bone cement were identified in 189 of the 500 specimens.
Double tetracycline labels indicating continuous bone turnover were identified in all biopsy specimens (Ste-Marie et al 2004).
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