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In contrast to MHCII+CD16- cells that generated very few TRAP+ cells on the bone wafer (data not shown), both MHCII+CD16int and MHCII+CD16+ cells were able to develop into mature OC- b)).
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The fluorochrome-labelled images confirmed the lack of difference in periosteal bone formation among these bones (data not shown).
β-AET did not significantly oppose these changes in cortical bone loss (data not shown).
We observed similar effects in a preventive model for breast cancer bone metastasis (data not shown).
We found no correlation between clinical outcome data and migration or bone loss (data not shown).
The use of corticosteroids or DMARD did not affect hand bone loss (data not shown).
Staining with control IgG did not show any immunoreactivity in the bone tissue (data not shown).
Direct measurements during surgery and bone wafer measurements do not reveal the true shape, provide rotational references, or maintain coordinate axes [ 10, 25].
Both cortical sites lose bone following ovariectomy and gain bone with teriparatide treatment (data not shown).
Metastasis to bone was not detected (data not shown).
Similar results were observed in bone-marrow derived dendritic cells (data not shown).
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