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No correlation between T or SHBG and BMD or markers of bone turnover could be found.
However, no correlation between E2 serum levels or FEI and bone density or markers of bone turnover could be observed.
During pregnancy, marked enhancement of bone turnover could be shown together with loss in BMD that is believed to be reversible [ 17].
The increase in bone turnover could be attributed to the enhanced osteoclastogenic activity induced by galectin-8 that subsequently promotes osteoblasts differentiation and increased BFR (Feng and McDonald, 2011; Bonucci and Ballanti, 2014).
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Although bone turnover markers could be assessed to confirm the changes noted on DXA scanning, we unfortunately do not have sufficient remaining serum or urine samples for this evaluation.
Second, active bone turnover could explain this phenomenon, such that hASC produced bone may have been gradually replaced overtime with mouse host derived tissue.
Measurement of markers of dynamic expression of bone turnover, enables evaluation of changes over time versus assessment of static changes in bone physiology that could be observed via DXA [ 50] is a conferred added strength.
No correlation between markers of bone turnover and sex steroids could be found.
Although, this study did not compare the inflammatory conditions of AD and AD, the improvement in bone turnover found in this study could be caused by the attenuation of the chronic inflammatory condition by AD.
Biochemical markers of bone turnover may be of value for prediction of individual bone loss.
Blood lead and bone turnover may be associated with the risk of amyotrophic lateral sclerosis (ALS).
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