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Skeletal fluorosis (with adverse changes in bone structure) may be observed when drinking water contains 3 6 mg of fluoride per liter, particularly with high water consumption.
Although MRI analyses did not reveal statistically significant abnormalities in the shape or size of skulls in Ate1-deficient mice (Fig. 8B, C), the MRI data did not preclude the possibility that bone structure may be perturbed in the absence of Ate1.
As a result of the balance between bone formation and resorption, bone structure may be modified and thus the biomechanical properties which are determinant of bone strength and fracture resistance.
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These relationships were not evident in trabecular bone from patients with OA, suggesting that bone regulatory processes leading to particular trabecular structures may be altered in this disease.
Crack-resistant engineering structures may be developed by mimicking bones.
Rammed earth structures may be long lasting.
CT currently provides quantitative measures of bone structure and may be used for estimating bone strength mathematically.
The effects of ZOL on the bone structure may or may not directly affect the progression of the cancer, but prevention of abnormal bone modeling in a tumour-ridden bone may be key to ZOL reducing the risk of skeletal related complications and prolonging the time to skeletal related event occurrence in patients with bone-metastatic prostate cancer [23], [24].
Alteration in subchondral bone remodelling, and subsequently in bone structure, may lead to changes in load distribution.
Considering that the three-dimensional structure of bone marrow may be important for the organization of specialized niches, undifferentiated (non-induced) BMSC were cultured on a non-adherent surface that favors cell aggregation.
The three dimensional structure of insoluble bone collagen may be stabilized similarly and made chemically less reactive than native collagen.
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