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In other words, bone strength may be weaker as normal turnover is inhibited.
Both type of drugs inhibit bone turnover, and thereby bone strength may be weaker as a result of treatment.
Moreover, animal studies indicate that even if BMD changes are smaller in response to PTH after pretreatment with a bisphosphonate, bone strength may be equally increased in the clinical setting as in the animals given PTH without prior antiresorptive agents (Ma et al 2003).
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Despite high systemic bone mineral density, bone quality and strength may be decreased at the systemic level in OA of the knee [ 12].
As a result of the lack of fatigue data on interdigitated cement, this interdigitated region was represented in the FEA model by material properties of bone cement, although its strength may be lower than that of pure bone cement (Race et al. 2003).
And their strength may be growing.
Jott's strength may be its convenience.
Majority strength may be dangerous.
Bone strength may also be compromised by changes in bone geometry or trabecular bone microarchitecture, which are not reflected in BMD measured with dual x-ray absorptiometry (DXA) [ 9- 11].
The increase in fracture risk associated with anastrozole was evident even at the first analysis, suggesting that the decline in bone strength may occur very rapidly in some patients, perhaps even before major changes in bone mineral density are detected.
Simple engineering analyses that estimate bone strength may yield overly optimistic results, a new study suggests.
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