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The performance of, and periprosthetic bone response to, a tapered, titanium (Ti6Al4V), hydroxyapatite-coated femoral hip prosthesis was evaluated at minimum of 10 years of follow-up.
Ashe et al. [ 18] reported in their cross-sectional study that the bone response to a nondominant fracture might differ from a dominant fracture.
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Eldecalcitol enhanced the cortical bone response to mechanical loading and a synergistic effect was observed in a rat model.
In a recent work, we evaluated the bone response to particulate debris in an ovariectomized mice model [ 6].
Eldecalcitol enhanced the cortical bone response to mechanical loading through a synergistic effect.
The aim of this study was to clarify the influence of eldecalcitol administration on bone response to mechanical loading using a four-point bending device.
In our work, densitometric and morphometric studies point to a better bone response to the Ti6Al4V commercial implants thermally treated at 700 °C for 1 h (Figs. 5 and 6).
The findings support the concept of osteoproliferation as being a local anabolic bone response to inflammation, mechanical stress, or microdamage, but not a systemic process in AS.
The present study was designed to evaluate the bone response to titanium implants treated with a thin Ca-P coating and bisphosphonate.
Therefore, the absence of a bone response to axial compression at the endocortical compartment could be attributed to the default of MSCs to differentiate into the osteoblastic lineage.
The cortical medial bone spike that may be seen in atypical femoral fractures is a focal bone response to the developing fracture.
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