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Modan-Moses D, Vered I, Schwartz G, et al. Peak bone mass in patients with phenylketonuria.
Human recombinant parathyroid hormone (1 34) (PTH) therapy increases bone mass in patients with osteoporosis.
The study has to be replicated in larger number of patients and is by no means standard treatment for low bone mass in patients with anorexia nervosa.
Bisphosphonates are a class of drugs that are widely used to inhibit loss of bone mass in patients.
It has also been shown to modestly increase bone mass in patients with established osteoporosis (Bonucci et al. 1995; Gennari et al. 1992; Reginster et al. 1994; de Vernejoul et al. 1990).
The efficacy of the signal to prevent and treat low bone mass in patients confined to bedrest, suffering from spinal cord injuries or osteoporosis, or inflicted with neuromuscular diseases will require further investigation.
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Although osteoclasts and osteoblasts are suspected to be involved in the low bone mass in IBD patients, the precise mechanisms of action remain unclear, especially whether activated T cells act on osteogenic progenitor cells is currently unknown.
Monitoring bone mass in dialysis patients is important.
Poorer bone mass in psychiatric patients has been reported in many literatures[ 9, 10, 23].
Many research projects have reported that hyperprolactinemia correlates with poor bone mass in schizophrenia patients.
Kalla and colleagues have also shown that DMARD treatment increase the cortical bone mass in RA patients [ 25].
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