Furthermore, culture medium obtained from the damaged osteocytes could induce TRACP-positive cells in bone marrow cell culture.
Mononuclear bone marrow cell (MN-BMC) transplantation has great clinical potential to promote myocardiogenesis and angiogenesis.
The most hydrophilic PEOT/PBT copolymers did not sustain goat bone marrow cell adhesion and growth.
Clinical studies on intracoronary bone marrow cell (BMCs) infusion in patients with acute myocardial infarction (AMI) revealed mixed results.
Combining molecular and biochemical analyses, it was found that bone marrow cell differentiation was stimulated over proliferation on GC-ICEL.
By the removal of haematoma tissue, essential factors for periosteal and bone marrow cell proliferation and differentiation and finally bone healing are severely reduced or withdrawn.
While these studies ruled out the scenario that bone marrow-derived cells directly contribute to nephrons, bone marrow cell contributions were confirmed in the renal interstitium [58, 59].
Furthermore, extracts from the membrane have been shown to stimulate bone marrow cell proliferation and differentiation of progenitor cells to the osteoblast lineage [4].
MTT assay was used for bone marrow cell lines (KG-1 and HBMF-SPH cell lines) to evaluate cytotoxicity of CLs and CL/miR-101.
Crack-free specimens of the bioglass ceramic were immersed in human bone marrow cell cultures for 3, 7, 14 and 21 days, in order to study biocompatibility.
Therapeutic angiogenesis was induced by local autologous bone marrow cell implantation (BMCI) in ischemic hindlimb or ischemic heart models in rats.
