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Exact(1)
This bone gain, which is attributed to the ability of cPTH to retain its capacity to stimulate bone formation in spite of diminished resorption, is in agreement with our previous report that cPTH effects on bone formation were preserved despite the absence of T cells [11].
Similar(59)
Bone formation was preserved in the osteolytic lesions induced by noggin-silenced PC-3 cells, suggesting that cancer cell-derived noggin interferes with physiologic bone coupling.
Bone formation is polarized towards the joint surface, and preserves a layer of articular cartilage [ 6].
Large osteophytes representing new bone formation are apparent at the joint margins (white arrows), and are associated with bone remodelling.
Bone formation was evaluated at 12 weeks.
At birth, bone formation is incomplete, and soft spots can be felt between these centres.
Orthotopic bone formation was compared to ectopic bone formation in comparable constructs.
Continuous bone formation is evident.
Evidence of new bone formation was prominent.
Bone formation was near complete suppression in the No Formation and Adynamic specimens.
Bone formation was substantial in the bone-derived mesenchymal matrix.
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