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Thus, pedomorphic bone development and changes in collagen gene expression patterns in pelagic notothenioid species could be due to one or more mutations affecting these regulatory genes.
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To appreciate why bone becomes weaker or less resilient to fracture with age in both men and women and in individuals of different races, a general knowledge of bone development and age-dependent changes is necessary.
During bone development and fracture healing, vascularization is observed before bone formation.
Bone development and remodelling requires a coordinated balance between bone-forming osteoblasts and bone-resorbing osteoclasts (Raggatt & Partridge, 2010).
Phosphorus intake is essential to normal bone development and mineralization.
Endochondral bone formation and chondrocyte hypertrophy are involved in bone development and longitudinal growth of long bones [ 20].
Alternatively, bone marrow derived macrophages can suppress erythroid lineage development and change properties.
Life is about development and change.
Thus, learning stands for development and change.
Mucoplysaccharidoses have a direct impact on cartilage and bone development resulting in advance degenerative changes in the spine (Fig. 32).
Substantial aluminum contamination may result in delayed bone development [31] and development of anemia.
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