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The risk of lower bone density was increased according to the increase in BCd levels after adjusting for eGFR and covariates, in which a significant interaction between BCd and eGFR existed.
At 3, 12 and 48 weeks of age bone density was increased in vertebral spine, but was, as shown before, statistically indistinguishable from wild type at 24 weeks of age ([12]; Figure 1B).
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However, bone density is increased rather than decreased in type 2 diabetes (3).
Bone mineral density was increased in IL-15R-deficient mice and was not reduced after ovariectomy.
Interestingly, however, when wild-type mice were infected with an avirulent Salmonella strain, which can induce OPG, osteoclast development was suppressed and bone mineral density was increased.
The present study manifests the following: in the 1 mm coronal bone defect area surrounding the implant, bone mass and density were increased merely by the SLA implant surface, but in the open bone defect area, 0.5 and 1.0 mg/mL concentrations of rhBMP-2 were more effective at promoting bone regeneration and osseointegration.
This increase in bone density was unexpected as the increased OC activity in Arf-/ mice would typically result in decreased bone density.
Increased bone density was demonstrated, and an uptake of the marker isotope in an epiphyseal plate by Gascintigram gradually emerged, suggesting that IL-6 or IL-6/sIL-6R complexes had previously interrupted the bone growth at epiphysis.
The increase in bone density was almost twice as great among women given the hormone substitute, although the two groups lost teeth at the same low rate.
A constant increase in bone density was used to model compaction since it was shown that compaction following insertion of a helical blade was independent of the initial bone mineral density.
Bone density was evaluated quantitatively.
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