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This indicated that the bone bridge may be resorbed by the distraction forces induced by the cartilage of the growth plate.
4. The formation of a bone bridge may be prevented by the transphyseal placement of a tendon graft. 5. Permanent transphyseal hardware placement can result in a growth abnormality.
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Small bone bridges may be resorbed without influencing the remaining growth process.
Roads and bridges may be boring, unsexy.
Bone bridge formation may be due to destruction of the epiphyseal circulation, but the mechanisms of bone bridge formation have not yet been clarified [1, 2].
The bone bridge formation may be detected approximately 4 weeks after injury by radiological or CT examination or by magnetic resonance [ 17, 18].
By impairment of enchondral ossification and normal chondrogenesis in the area of growth cartilage of the long bones of the extremities, a formation of the bone bridge may occur with a subsequent disturbance of bone growth [ 1, 2].
3. Small bone bridges may resolve spontaneously.
For example Schmidt et al. [15] and Schieker et al. [33] proposed 3 cm as the critical size, while other authors judged 4 cm to be the ultimate bone defect that may be bridged by a cancellous bone graft [34].
In these cases, bridging therapy may be used.
Bone bridges are expected to be evident after RFA epiphysiodesis.
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