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In osteochondral lesions, subchondral bone appears to require longer periods of time than cartilage for remodeling.
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With respect to bone preservation in rodents, DHEA was thought to act directly via a DHEA-specific receptor [9], while this same activity (bone preservation) appears to require conversion to sex steroids in humans [14].
Prosthetic migration appears to require a fibrous tissue at the interface between prosthesis and bone.
Acceptance of such extraordinary findings appears to require extraordinary evidence.
This slow signal appears to require some time to change.
Percutaneous cryoablation appears to require less analgesia than RFA [ 36].
Thus, neurotoxicity appears to require toxic oligomeric assemblies of Aβ.
The mechanisms whereby testosterone affects muscle, bone, and the erythron do not appear to require prior molecular conversion [ 30].
hL-PYK activity does not appear to require monovalent cation.
The bone appears to have been first roughly shaped with a stone blade.
Subchondral OA bone appears to be a relevant therapeutic target.
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