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Our lectin blot experiments demonstrated that numerous proteins in choriocarcinoma exhibit β1-4GlcNAc glycosylation, which were approximately 40 150 kDa.
Southern blot experiments demonstrated that the abundance of actin gene and of the abundant spike mRNA SRG3 have been greatly reduced by the normalisation process (data not shown).
Northern blot experiments demonstrated that chicken liver and intestine express the highest levels of the specific transcript with a size of approximately 830 nt.
Similarly, western blot experiments demonstrated that the protein levels of these genes were significantly suppressed to various degrees by miR-188 overexpression in both transiently and stably transfected cells.
Western blot experiments demonstrated that HOCl LDL led to activation of procaspases-3 (after 8 h) and PARP cleavage in a time-dependent manner starting 4 h after treatment (Fig. 1B).
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Western blotting experiments demonstrated that BMP-6 treatment obviously inhibited both c-Fos and c-Jun expression (Figure 4D).
Because Northern blot experiments demonstrated a number of different FGFR2 isoforms, Western blot analysis was performed to examine the number and level of expression of FGFR2 protein(s).
Accordingly, indirect immunofluoresence, as well as western blot analysis experiments demonstrated that this "induction" of a proliferative phenotype was accompanied by a significant decrease in the expression of the contractile phenotype markers α-actin, SM-MHC and desmin.
In addition, western blot analysis from three independent experiments demonstrated that expression of TERT did not reduce progerin levels.
43 Analysis of stimulated protein kinase C isoenzyme expression, Western blot protein studies, and siRNA knockdown experiments demonstrated that activation of PKCδ in endothelial cells is responsible for the direct effects of ingenol mebutate on endothelial cells that contribute to neutrophil recruitment.
Western blot and qRT-PCR (quantitative real-time PCR) experiments demonstrated that compound 6r may induce apoptosis through both of intrinsic and extrinsic apoptosis pathway.
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