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The latter blocker did not affect single channel characteristics of Mrs2.
The CCD blocker did not affect test results where CCDs were not involved.
In contrast to TEA-Cl, verapamil, the Ca2+ channel blocker, did not affect IAA- and FC-induced growth.
On the other hand, TEA (tetraethylammonium), a nonspecific K+ channel blocker, did not affect the vasodilatory activity of -praeruptorin A against PE-induced contraction.
Pretreatment of cells with 5 mM probenecid, a Panx1 hemichannel blocker, did not affect the spread of dye into the vacuole, suggesting that Panx1-formed hemichannels are not involved in this phenomenon [ 103].
Likewise, Pyr 6 (10 μM, 5 min), a recently synthesized specific Orai1 blocker did not affect the biphasic Ca2+ response to CPA (n = 82; Figures 4(g)- 4(h)).
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In contrast, in the same animal model, it was shown that nisoxitine, a norepinephrine transporter blocker, does not affect locomotor hyperactivity.
The blockers did not affect the AtoS∼P to AtoC phosphotransfer.
The inclusion in the latter models of dichotomous variables representing current active treatment with common anti-hypertensive drug classes (i.e. ACE-I/ARBs, β-blockers, calcium channel blockers) did not affect the results.
Treatment with β-Blockers, aspirin, ACE Inhibitors and Ca2+ Channel Blockers did not affect the adipocytokine gene expression in the epicardial adipose tissue (data not shown).
We find that Cl− channel blockers do not affect PS exposure in response to a Ca2+ ionophore, implying that Cl− conductances are not necessary for platelet PS exposure.
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CEO of Professional Science Editing for Scientists @ prosciediting.com