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Furthermore, excessive CC49 antibody successfully blocked the binding of CC49-QDs to the MGC80-3 cells, indicating that the binding was mediated through TAG-72.
KYK-2.0 Fab blocked the binding of the natural human NKG2D ligands MICA, MICB, and ULBP2 as potently as a commercially available mouse anti-human NKG2D monoclonal antibody in immunoglobulin G (IgG) format.
The same concentration of sCD4 blocked the binding of MAb b12 by 95% (Figure 9A).
In addition, SSL5 blocked the binding of anti-PSGL-1 mAb KPL-1 to HL60 cells (data not shown).
Furthermore, pre-incubation of CD2-BV-coated wells with anti-CD2 antibody blocked the binding of CD58-BV (Fig. 2B right panel).
To examine these possibilities, gp120 was first incubated with sCD4, which at 10 nM blocked the binding of anti-MCP sera or MAb 11A8 by 50%.
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To block the binding of the endogenous Sema3 with Nrps, we used function-blocking antibodies that specifically blocked ligand-binding sites in Nrps.
They exert their action by blocking the binding of AngII on the AT1 receptor.
Blocking the binding of VCAM to α4β1 is thought to be a way of controlling a number of disease processes.
When there is no peptide binding, the side chain of W65 is rotated toward the center of the aromatic cage and block the binding of trimethylated histone peptides.
Therefore, it appears impossible to block the binding of HosFn to mouse TfR1 with the antibody used in Chen et al.'s paper.
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