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As control, an untreated PGS block was implanted into the contralateral defect (BMP group).
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HAp/PLLA blocks were implanted into mice (intraperitoneally) and after implantation, the phenomenon was studied histopathologically and by FT-IR spectroscopy.
A second block was implanted onto the contralateral muscle after 1, 2 or 4 weeks and 200 μg rhBMP-2 was injected into the blocks on both sides.
A PGS block soaked in rhBMP-2 (400 μg/ml) was implanted into one defect (BMP group).
Either a gelatin-coated or uncoated β-TCP block (samples E and O in Table 1) was implanted into the defect, and the skin was sutured.
All cell lines were implanted into C57BL/6 mice, except 4T1, which was implanted into BalbC mice.
(E) An ACS was implanted into the bone defect.
This was implanted into the mother and the baby was born nine months later.
The gene was implanted into bacteria, which could then make insulin.
was implanted into the bag.
The PEEK implant, A-TiO2/PEEK implant or R-TiO2/PEEK implant, was implanted into a distal femur in each rabbit.
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