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Our analysis suggests that low birth weight does not itself explain the increased metabolic risk associated with birth order.
This suggests that birth weight does not fully mediate the association between maternal glucose levels and offspring adiposity.
We may conclude, therefore, that low birth weight does not explain the observed association between parental smoking and hepatoblastoma.
Certainly a short mother with estimated low birth weight does not carry a high risk for CPD.
Birth weight does not reflect the complete picture of body morphology such as body lengths or circumferences.
However, many observational studies have shown that additional adjustment for birth weight does not explain the observed associations [ 38].
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Recent studies have shown that poverty, education, access to prenatal care, smoking and even low birth weight do not alone explain the racial gap in infant mortality, and that even black women with graduate degrees are more likely to lose a child in its first year than are white women who did not finish high school.
But the risk of low birth weight did not differ significantly between the three treatment groups across gravidity (Table 4).
However, adjusting survival traits for birth weight did not result in major differences in the results of association of PrP genotypes with postnatal lamb survival (Figure S2).
The risk of third trimester severe anaemia or low birth weight did not differ significantly between the three treatment groups regardless of gravidity.
Similarly, the risk of low birth weight did not differ significantly between the three treatment groups regardless of gravidity; (RD = -1.17 [95% CI; -4.39-1.02] for IST-SP vs. SP-IPTp and RD = 0.78 [95% CI; -2.11-3.68] for IST-AQAS vs. SP-IPTp).
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