Sentence examples for birth data not shown from inspiring English sources

Most of the mutants initiated growth defects beginning around 7 to 10 days after birth (data not shown).

From the first comprehensive model of NGF decline from birth [2] we can calculate that the maximum NGF recruitment occurs at birth (data not shown).

These Cdk4-null pituitaries display normal Ki67 staining during embryonic development but a decreased proliferation after birth (data not shown), similarly to that we have previously observed in Cdk4-null endocrine cells in the pancreas [49].

Analysis of boys who remained cryptorchid at 3 months of age showed comparable results to boys with cryptorchidism at birth (data not shown), which however, did not reach statistical significance due to the small group size (n = 33).

The hair cells around the vesicles could be detected at least until P30 (P16 shown in Fig. 1K K") and the vesicles inside the ganglia persisted for about 9 month after birth (data not shown).

Ap2aIREScre/+; COET animals did not survive after birth (data not shown) and examination of E18.5 embryos revealed bilateral cleft lip (Figure 4a a') and cardiac outflow tract (OFT) defects (Figure 4b b') in all animals examined (n>20), and control animals exhibited a normal phenotype (Figure 4a and b, and data not shown).

As expected (based on our analysis of Snap23fl/wt mice), Snap23Δ/Δ pups were never obtained from adult heterozygous matings after more than 50 live births (data not shown).

Similar associations were also observed when the analysis was restricted to only live births (data not shown).

We found, as expected (Trichopoulos et al, 1983; Rosner et al, 1994), a gradual increase in breast cancer risk with increasing age at first birth (Table 1-wrap>), and an additional protection with increasing number of births (data not shown).

Similar results were found when comparing those with low birth weight (birth weight <2.5 kg) to those with normal birth weight (data not shown).

The independence of the observed associations from birth weight, coupled with the absence of direct associations of the haplotypes and htSNP in question with birth weight (data not shown), suggests that the effects of the risk haplotypes and alleles on childhood ALL risk are not directly mediated by birth weight.