Both psychosocial and biochemical mechanisms can be causative factors in depression.
Thus biochemical mechanisms can be analyzed for their potential for some instability in an efficient way.
These key, sensitivity analysis identified biochemical mechanisms can be matched to drugs known to perturb the identified mechanisms, yielding a ranked list of potential drugs.
Mathematical modeling in terms of fundamental physical and biochemical mechanisms can be used to justify experimental results and determine future research directions [ 110– 110].
A range of studies have described the biochemical mechanisms in the development and progression of diabetic retinopathy; however, no mechanism can be regarded as established.
The mechanism can be employed as the reception mechanism in nanoreceivers and modeled as a biochemical reaction.
Mass flow and biochemical or genetic regulation mechanisms can be equally represented at arbitrary abstraction level, ranging from molecular via cellular level [5] [7] to multi-cellular level, describing, e.g., developmental processes in multi-cellular pattern formation [8], [9].
Dissection of Ca2+ signaling specificity mechanisms can be advanced through characterization of the combined cellular, genetic, and biochemical mechanisms in a single cell type.
However, two distinct mechanisms can be envisaged.
The effects of physical activity on depression mechanisms can be explained with both psychological and physiological/biochemical theories.
