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Biochemical analyses found that these acellular tissues with distinct porous structures consisted primarily of insoluble collagen, elastin, and tightly bound glycosaminoglycans.
Early biochemical analyses found that exogenously expressed murine MELK binds to the zinc-finger-like Zpr9, which results in the activation of the oncogenic transcription factor B-Myb in murine cell lines [38]. Figure 2 This schematic highlights many of the signaling pathways MELK is involved in.
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A combination of physiological, biochemical and genetic analyses found that AtOCD does affect proline accumulation at low water potential via a mechanism that is independent of P5CS1.
Subsequent analyses found that nearly all the interceptions had failed.
Combining molecular and biochemical analyses, it was found that bone marrow cell differentiation was stimulated over proliferation on GC-ICEL.
Using a biochemical approach, we found that MYOC localizes intracellularly to both the cytosolic and particulate fractions.
Importantly, we find that DOA disease alleles disrupt key biochemical features of OPA1.
We find that CARS imaging gives quantification similar to standard biochemical triglyceride quantification.
Using X-ray crystallographic, biochemical, and genetic analyses, we find that the monovalent cation-binding site is conserved in MutL, but both Na+ and K+ support the MutL ATPase activity.
Computer-assisted and biochemical analyses suggest that there is a complex secondary structure in this region that is comparable to the secondary structures that are found in picornaviruses (E.A. Brown, H. Zhang, L.-H. L.-H. PingS.M. Lemon, Nucleic anddS.M.s. 20:5041-5045, 1992).
Structural and biochemical analyses demonstrated that H3mm7 nucleosomes are unstable without posttranscriptional modification.
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