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No differences in initial binding were observed at 5 µl/min or above indicating no limitations to any combinations.
Finally, low levels of PU.1 binding were observed at the Flt3 locus.
No correlation was observed with fractional anisotropy at the FWE corrected threshold P < 0.05, but trends toward negative correlations between fractional anisotropy and action binding were observed at P < 0.1, FWE corrected, in the anterior corpus callosum and prefrontal white matter tracts similar to those identified from mean diffusivity (Supplementary Fig. 2).
Similar(57)
Consistent with this, a version of the CaM-binding site peptide in which the equivalent of Ser has been replaced by a phosphoserine did not bind at all to CaM at pH 7.5, although some binding was observed at pH 6.5.
However, a 50% reduction of BRCA1 230-534 binding is observed at a concentration where p53 is unable to bind (compare lane 4 and 8 in figure 5c).
The highest incidence of Ca2+ and Na+ binding is observed at a highly acidic pocket on the protein surface.
No COUP-TFI binding was observed at the negative control genomic region (the housekeeping gene Cyclophilin A) (Figure 4A).
As expected, strongest binding is observed at physiological pH.
No correlation between tau pathology and [H]AZD2995 or [H]AZD2184 binding was observed at 1 nM (Fig. 4d).
Specific CHD7 binding was observed at three Otx2 enhancer elements identified by Kurokawa et al. (Kurokawa et al., 2004a, 2004b).
At limiting CEACAM1 concentration, decreased CEACAM1 binding was observed at one or more UspA1 concentrations for all of the other constructs.
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