Sentence examples for binding were demonstrated in from inspiring English sources

Agonist induced changes in GTPγS binding were demonstrated in fused Gsα/β2-adrenoceptors where agonist efficacy was well reflected by changes in the kinetics of GTPγS binding (Wenzel-Seifert and Seifert, 2000; Seifert et al., 2001).

Not only can binding be demonstrated in vitro, the two proteins also co-localize in activated cells at the cortex.

Specific binding of 111In-DTPA-cNGR was confirmed in the Matrigel model and, moreover, binding was demonstrated in the infarcted myocardium and infarct border zone.

Estradiol binding was demonstrated in MCF-10A cell membrane fractions and may explain the estradiol action in these cells that lack intracellular ER.

No binding was demonstrated in CAIX negative CaKi 2 and HUVEC cells.

The specificity of the [35S]GTPγS binding is demonstrated in Fig. 7 (C, F, I) where an excess of cold GTPγS was included in the incubation.

Significant glucose-fiber binding was demonstrated in the in-vitro models.

Increased C-DED binding was demonstrated in the brain of patients with Creutzfeldt-Jacob disease [ 67].

Specific low affinity (Kd = 166 nM), high capacity (Bmax = 1.2 pmol mg-1 protein) somatostatin binding was demonstrated in 22 of the gastric cancers and 17 of the colorectal cancers (Kd = 140 nM, Bmax = 1.8 pmol mg-1 protein).

The binding models were demonstrated in the aspects of inhibitor's conformation, subsite interaction, and hydrogen bonding.

Similar binding effects were demonstrated in Hep3B-sh1 and Huh-7 cells (data not shown).