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Exact(16)
As expected, displacement of 18F-nifene binding was seen in the post-nicotine challenge similar to that reported for 2-[18F]F-A-85380 [17].
The results showed significant binding of the anti-gp41 antibody to the Ad5/HVR2-MPER-L15(Gag) and Ad5/HVR2-MPER-L15ΔE1, whereas no binding was seen in response to AdCMVGag control.
A significant reduction in enzyme activity was seen in the F46Y and L172Q variants and a significant increase in substrate binding was seen in V38A, L172Q, K185E, and R258C[4].
As no inhibition of IgE binding was seen in our experiments using anti-CD23 antibodies, the higher affinity of FcεRI to the Fc domain of IgE [10] could possibly enhance IgE transport.
Binding of ERG to the EZH2 promoter was observed in ERG translocation positive VCaP cells and in LNCaP and 22Rv1 cells upon stable expression of ERG, while no binding was seen in non-ERG expressing parental LNCaP and 22Rv1 cells (Fig. 3F).
No AQP4 binding was seen in rats receiving control IgG.
Similar(44)
Elevations in dopamine D3 receptor mRNA and binding are seen in the denervated striatum of hemiparkinsonian rats treated chronically with levodopa, and these changes correlate well with behavioural sensitization in this model.
Maximum binding was found in the thalamus, while moderate binding is seen in the cortex, and minimal binding in the cerebellum.
Visually, this study looks normal, although a mild reduction in DAT binding is seen in the left posterior putamen and possibly on the right posterior putamen as seen in the ACSC reconstruction.
No significant alterations in DAT binding were seen in the 1∶10 A53T α-syn or in either concentration of GFP.
Finally, in wildtype animals a substantial decrease in p50 DNA binding is seen in aged tissue compared to young.
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