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As shown in Fig. 7a, MHC I expression and BNP Ab binding was much lower on endothelial cells than on PBMC.
The expression level of E2F (related to cell division) in rad mutant was clearly lower than WT at one and three days, and that of Zinc fingers (related to protein binding) was much lower at 1 day (Fig. 1).
Staurosporine also induced a significant (p < 0.01) increase in caspase labelling within 2 h of treatment as compared to mock-treated controls (Fig 6a, b); however, inhibitor binding was much lower when compared to that observed in response to miltefosine and reached control levels already at 4 h of treatment.
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Furthermore, SHP2-binding activity of v225d CagA was much lower than that of CagA of H. pylori isolated from Western countries (Western CagA).
The binding contact of FetA for enterobactin was much lower than that for other enterobactin receptors, and it was therefore proposed that this receptor could interact with high affinity to an as-yet unidentified phenolate siderophore.
In the present work, we found that the Clerodendrum polysaccharide sub-fraction CSP-AU1 induced secretion of TNF in human PBMCs mainly via TLR4, but binding affinity for this TLR agonist was much lower than that of E.coli-derived LPS.
The tumour uptake of the 99mTc-HYNIC-PEG4-dimer was much lower, suggesting monovalent binding.
Notably, binding of the two-domain fragment to β7 was much lower.
The receptor number for the A375-SM cell line was much lower and not statistically different to the level of nonspecific binding achieved.
The binding of PA to the GNA, DSA, and PNA specific for Man, Gal and GlcNAc was much lower.
Unemployment was much lower than now.
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