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Maximum binding was found with bacteria in stationary phase.
No binding was found with the other dysferlin C2 domains in either the presence or absence of Ca2+ (Fig. 5A).
Regarding SNP-638, non-specific protein binding was found with a −638G probe (lane 7th), which was evenly reduced by a cold competitor with G at −628 (8th lane) and that with A at −638 (9th lane).
The maximum binding was found with 40 and 80 ng proteins.
Enhanced cell death induction upon CD70 binding was found with the TRAILR1- as well as with the TRAILR2-preference variant of scFv l αhCD70-TNC-TRAIL.
No binding was found with lentil lectin or concanavalin A. Antigenic activity was strongly reduced by trypsin and subtilysin digestion and by treatment with sodium periodate, but it was not affected by neuraminidase.
Similar(54)
β-OG ligand binding was found to vary with respect to hydrophobicity, hydrophilicity, hydrogen bonding, van der Waals interactions with ligands and tightness of the binding site.
In particular, radioligand binding was found to increase with increasing cell number in the 6-well plate (p<0.05) (Figure 4B).
Mu-opioid receptor binding was found to increase with age in neocortical areas and the putamen.
Specifically, an antibody inhibiting VEGF-NRP1 binding was found to interfere with retinal vascular remodeling as well as tumor angiogenesis (Pan et al., 2007) and is currently being developed as a therapeutic strategy to block vessel outgrowth.
Erythrocyte anionic charge by itself is unlikely to be important in the pathogenesis of diabetic retinopathy but reduced RBC alcian blue binding was found to be associated with the loss of glomerular basement membrane anionic charges in diabetic rats and patients [ 19].
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