Sentence examples for binding was demonstrated to from inspiring English sources

Binding of antibodies specific for clone A12.2 was abolished when trimeric gp160 was denatured (Figure 3, field A1 and B1), although specific binding was demonstrated to native Env.

However, in a chromatin immuno-precipitation (ChIP) study preformed by Scherf and colleagues [24] using anti-pfsir2 antibodies, pfsir2 binding was demonstrated to occur in telomeric regions with repression of upsB expression while it was also clearly shown to not bind or have a direct effect on upsC var genes (upsA genes were not studied).

In Japan, C. jejuni ST-4526 host cell binding was demonstrated to be increased for FHA-positive strains compared with FHA-truncated strains (Asakura et al. 2012).

As expected, reproducible binding was demonstrated to promoters of known p53-inducible target genes shown in Figures 4 and 5 including p21Waf1/Cip1, MDM2 and cyclin G1.

In a previous work, the S. oneidensis so2426 gene, annotated as a DNA-binding response regulator, was demonstrated to be specifically responsive at both the transcript and protein levels to acute chromate [Cr VI)] challenge.

Indeed, the Aly/REF-binding motif of EB2 was demonstrated to be essential both for its mRNA export function and for production of infectious virions [28].

In vitro and in vivo binding of ALG-2 to Scotin was demonstrated to be strictly calcium dependent indicating a role of this interaction in calcium signaling pathways.

JA-Ile was demonstrated to mediate binding of the co-receptor proteins CORONATINE INSENSITIVE1 (COI1) and JASMONATE ZIM DOMAIN (JAZ), thereby triggering JA responses [ 14– 17].

Control of enzymatic activity and binding was demonstrated on substrate emulsions and natural lipid layers.

Selective binding was demonstrated by turbidity assays with Ricinus Communis Agglutinin (RCA120) lectin.

No binding was demonstrated in CAIX negative CaKi 2 and HUVEC cells.