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In our earlier study the dramatic decrease in the CLA of PDC-109 upon PrC binding was attributed to the dissociation of the polydisperse aggregates of the protein [23].
This also corroborates our earlier finding [ 59, 60, 63] that phosphorylation of T18 does not diminish helicity of TA to the extent that it should disrupt binding to MDM2; indeed the disruption of binding was attributed to the development of electrostatic repulsions between phosphorylated T18 and the MDM2 surface and our current results are in accord with our earlier hypothesis [ 17].
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Based on the observed binding capacity, the binding molar ratio between HIgG and MEL-A is approximately 1 70, supporting the binding is attributed to the "multivalent effect".
In Gly48Val, loss of SQV binding is attributed to the loss of a hydrogen bond between the carbonyl oxygen of 48 and the amide of SQV.
It was previously reported that truncated RyR1 without the C-terminal 15 amino acids lacks high-affinity ryanodine binding that was attributed to impaired tetrameric assembly [ 9], although it is possible that the deletion could have directly affected the ryanodine-binding site.
Compared with wild-type control, the binding of Tbx3 did not show significant change in Tbx3 HET mice, indicating that decreased binding of Tbx3 was attributed to a decrease of Baf250a protein.
The sensitivity improvement was attributed to binding of OH− ions to edge defect sites, but the binding characteristic was not thoroughly determined.
The decrease in swelling was attributed to the binding of nHaP to the chitosan and gelatin network.
The difference was attributed to the binding efficiency between the numbers of accessible streptavidin or carboxyl groups on the Qdots.
An increase in adsorption capacity was attributed to cadmium binding affinity of sulfur atoms due to soft acid base reaction and supported by a − ΔG value [26].
Aggregation of the PVP coated AgNPs (PVP-AgNPs) was enhanced by cysteine addition at high ionic strengths, which was attributed to cysteine binding to the AgNPs and replacing the otherwise steric stabilizing agent PVP.
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