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No reduction in binding was also noted for V39A, confirming UspA1 binds to the monomeric form of CEACAM1 as this mutation has previously been shown to abolish dimerisation (Watt et al, 2001).
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Transcription factor binding site enrichment was also noted.
An appreciable reduction in the plasma binding of MTX was also noted in vivo when KP was associated.
An even higher increase (27-fold) of RARβ binding to the CYP26A1 promoter was also noted.
In addition, constitutive Sp1 binding to the MMP-14 promoter was also noted in rat glomerular mesangial cells [ 45].
It was also noted that the binding force between CD36 and IRBCs decreased by a level of 30-50% after free CIDR was added.
It was also noted that non-specific binding in nocodazole-treated cells was higher than in DMSO-treated or untreated cells.
The participant-preferred socket was also noted.
A remyelination process was also noted.
Attenborough was also noted as a director.
Fibroplasia was also noted.
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