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The calcium binding was also increased in collagen exposed to glucose.
Since the HEK-293T cell line had endogenous TβRII, TGF-β1 binding was also increased dose-dependently in control HEK-293T cells.
P-H3 binding was also increased, showing that this nucleosomal response is involved, at least in part, in chromatin remodeling on the PGC-1α promoter.
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One of these genes, takeout, thought to be involved in circadian rhythms, feeding behavior and juvenile hormone binding is also increased in four other life span extending conditions: Rpd3, Indy, chico and methuselah.
NFκB nuclear binding activity was also increased by NO.
NF-κB binding activity was also increased markedly.
The NF-κB-binding activity was also increased, but there were no significant differences between the mice ventilated with room air and hyperoxia.
Similarly, the amount of p27 binding to nuclear Cdk2 was also increased in NMuMG cells treated with TGF-β1 + rapamycin.
H4K5Ac binding around Crem TSS was also increased after the acute METH administration in METH-naïve rats but not in METH-pretreated rats.
Although not statistically significant, STAT3 binding at this site was also increased by PRL by approximately 2-fold, an effect that was abrogated by pretreatment with WP1066 but not the STAT5 inhibitor.
The DNA-binding activity of LXRα was also increased (Fig. 1E).
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