Sentence examples for binding was accounted for from inspiring English sources

Of this, only < 6.8% of the binding was accounted for by binding to albumin (43 mg/mL) under these conditions.

The binding energies were in the range of −192 to −100 kJ/mol for the Prelog complexes (−105 to −29 kJ/mol total binding energy (TBE) if binding entropy was accounted for), which were approximately 34 kJ/mol lower than those of the accessible anti-Prelog complexes.

We observed a small amount of non-specific binding, which was accounted for in the densitometry calculations.

Strong support for the central role of non-nuclear antigen-binding auto-antibodies in the pathogenesis of LN can be found in the seminal observation that less than 10% of the total IgG eluted from kidneys of LN patients was accounted for by antibodies binding to dsDNA, C1q, Sm, SSA (Sjögren syndrome antigen A), SSB, histone, and chromatin [ 6].

It was accounted for".

Thus the absence of activation of monomeric IDE in the presence of binding can be accounted for by the necessity of a conformational change induced by activator binding to one subunit and transmitted to the adjacent subunit.

Because the amount of functional NTS1 in the SN fraction is reduced by 9% over the 24 hour time period needed for completion of solubilization and the IMAC step (Table 1), almost all [3H]NT binding sites are accounted for, suggesting that immobilization per se of NTS1 via the C-terminal histidine tag does not result in substantial receptor destabilization.

The amphipathic nature of VS toxins is consistent with the headgroup/tail interface of lipid bilayers, and it has been suggested that a significant fraction of the apparent free energy of VS/toxin binding could be accounted for by the free energy of toxin partitioning into the membrane (5), especially if the VS/toxin interface consists of only several residue pairs (2).

For all other peptides, the relationship between their T cell activation potency and TCR binding affinity is accounted for by the stability of the pMHC complex; e.g. A3V is a relatively weak agonist due to the poor stability of the pMHC complex (<1h), whereas M9C is a potent agonist due to its much higher affinity for H-2Db [ 18] causing a greater stability of its pMHC complex (>1h).

These numbers also indicate that there is a large amount of specificity in Ubx binding that cannot be accounted for simply by its preference to bind its monomer binding site, TAAT[t/g]G.

This leaves only ~17 kJ/mol of binding energy to be accounted for.