Sentence examples for binding to these complexes from inspiring English sources

Both of these complexes have a similar association rate for RNA, but a very much slower dissociation rate than that seen for the eIF4AIII-MLN51 SELOR complex in the absence of a nucleotide, showing that RNA binding to these complexes is much tighter.

Inhibitors of cyclin/Cdk complexes, by binding to these complexes, negatively regulate cell cycle progression [ 2].

LPS binding to these complexes facilitates activation of the TLR4/nuclear factor (NF -κB iNF -κBatory pathway, ultinflammatorying to the pathwayion of proatherogenic cytokines inclultimatelyr necrosis factor aleadingNF-α), intorleukin-6 (IL-6), and IL-1 [ 8- 11].

Antibodies in the form of immune complexes might play a central role in triggering inflammatory pathways in the joint [18], especially C5a binding to these immune complexes can attract granulocytes to the articular cartilage that can release inflammatory mediators (proteases, cytokines, chemokines, and reactive oxygen and nitrogen radicals) perpetuating inflammation and autoimmunity.

Furthermore, the cells binding to the complexes can be easily distinguished in CT images.

They identified approximately 8% of the human embryonic genome as binding to polycomb complexes and these genes have been designated as putative polycomb targets.

Because of the very high sensitivity of these spectra to binding to protein complexes such as SCΔ60, particularly for flexible peptides such as CpxI(26-48), even a small pevenntage of binding smalld be reflected in some cross-percentageening.

(B ) Sly1 binding to Golgi SNARE complexes (S.C).

Services allow dynamic binding to accomplish complex tasks for a given client.

The parameters for forming the effector protein-chaperone complexes, for binding of these complexes to the TTSS, for the probability that binding leads to "injection" and for the time required for "injection" could be adjusted freely (Fig. 3A; see also supplemental online materials).

Furthermore, the binding of complement component C1q to these complexes may explain the production of anti-C1q antibodies, which is found in approximately one-third of patients with SLE [ 3].