Sentence examples for binding to the seed from inspiring English sources
Based on our experimental findings and relevant literature, we synthesize a possible mechanism for arsenic binding to the seed biosorbent.
To confirm that the reduction in red fluorescence observed following overexpression of miR-212 is mediated through the predicted target site, we tested whether this effect could be prevented by mutating the sequence of the site to disrupt binding to the miR-212 seed region.
Twenty-four specimenscimens with relatively high IgE-binding to the rice seed albumin/globulin fraction were selected as rice-positive specimens, whereas three specimens with relatively low IgE-binding were selected as control specimens.
These have focused on the 5′ "seed" region of miRNAs binding to the 3′ untranslated region of targeted mRNAs.
Furthermore, when the predicted targeting site in hTERT 3′ UTR was mutated to avoid binding to the 'seed region', miR-1207-5p and miR-1266 were not able to effectively suppress the luciferase expression under the control of the hTERT 3′ UTR.
In order to confirm this and to determine the structural features required for binding, the inhibition of LM15 binding to tamarind seed xyloglucan by the presence of a range of xyloglucan-derived and related oligosaccharides was determined using competitive-inhibition (hapten) ELISAs.
These results indicate that suppression is due to direct miR-125b binding to complementary seed sequence in ADAMTS-4 3'-UTR.
Figure 5 IgE binding to seed extracts of rice lines with reduced levels of the major and the HMW allergens.
The best studied non-coding RNAs are microRNAs (miRNAs), 18 25 nt-long small RNA that epigenetically regulate translation by binding to a complementary "seed" sequence common to their "target" mRNA [2].
These small noncoding RNAs are able to control mRNA decay and protein translation by binding to seed sequences within target mRNAs (20).
Mutual conformational selection and population shift followed by minor induced-fit optimization is the key mechanism in biomolecular recognition, and monomers and small oligomers binding to amyloid seeds in fibril growth is a molecular recognition event.
