Sentence examples for binding to human endothelial from inspiring English sources

The cell-specific peptide TPS (TPSLEQRTVYAK) has been proposed as a potential candidate for fabricating tissue engineering scaffolds based on its ability of binding to human endothelial progenitor cells (EPC) with high affinity and specificity.

The structural determinant information was then applied to designing cystine-stabilized miniproteins binding to human vascular endothelial growth factor.

However, these parasitized CB RBCs are impaired in their binding to human microvascular endothelial cells (MVECs), monocytes, and nonparasitized RBCs – cytoadherence interactions that have been implicated in the development of high parasite densities and the symptoms of malaria.

Annexin V binding to human umbilical vein endothelial cells was assessed by flow cytometry after 24-hour culture with plasma.

Depletion of IgG from sera with high capacity to inhibit binding of annexin V induced a 2.7-fold increase in binding and preincubation of sera with cardiolipin and phosphorylcholine resulted in increase of median fluorescence intensity of annexin V binding to human umbilical vein endothelial cells.

Using this strategy, new peptide ligands were identified that showed significant binding to human brain endothelium but not to other human endothelial cells, so they may be used for specific targeting of drugs to the blood-brain barrier [ 171].

We also observed low signals of binding to human siglec-2 (B cells), L-sectin (sinusoidal endothelial cells), and human L-selectin (lymphocytes) for Pb01.

However, there are limitations to studying human endothelial function in vivo, or human vascular segments ex vivo.

We have synthesized and characterized polypyrrole/sodium nitrate (PPY/NaNO3) membranes that can be used as substrate to support human endothelial cell proliferation.

This result provides an important lead toward the development of high-affinity inhibitors of Ebola virus binding to sinusoidal endothelial cells as well as confirming that studies in the mouse will be relevant to humans as well.

We found that HbF impairs the binding of parasitized RBCs to human microvascular endothelial cells (MVECs), monocytes, and nonparasitized erythrocytes – cytoadherence interactions that contribute to the development of high parasite densities and the symptoms of malaria.