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While highly conserved regions as primer binding sites were avoided to prevent amplification of human DNA, less conserved regions as primer-binding sites may explain the lack of detection of certain mucorales by the ITS-2 assay.
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Calloused sites were avoided.
To avoid the possibility of the gfp-TALEN−induced mutation of yfp, its binding sites were modified by introducing synonymous codons, leaving the polypeptide sequence unaltered.
Potential binding sites were elucidated by docking.
Ligand binding sites were predicted using 3DLigandSite.
Two binding sites were identified for GF.
The residues contacted by US2 are located in a region between the C-terminus of the α2 helix and the α3 domain and are therefore distant from PLC contact sites on MHC I. Hence, binding site competition is avoided and allows US2 to attack MHC I molecules that are already associated with the PLC.
The low odds ratios near genes are likely due to the fact that ETSwt/CHD1del prostate cancers avoid breakpoints near genes while many protein binding sites are fixed near gene promoters.
Here, these binding sites are not discussed one by one.
One method to reduce the presence of these information-poor binding sites is to apply stricter binding site conservation criteria.
These binding sites are highly charged.
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