It should be noted that the MyTH4 FERM cassette may possess uncovered binding sites for these binding partners or that these bind other domains of myosin-X.
The structures indicate that CusB consists of multiple binding sites for these metal ions.
We consider a set of miRNAs known to regulate PTEN and identify high-confidence binding sites for these miRNAs on the 3′ UTR of protein coding genes.
These complexes were also subjected to MDS, which helped in predicting the energetically favorable binding sites for these ligands.
Findings show that the binding sites for these modulators are found in the amino terminal domain of such receptors, but different modulators appear to affect different subunits.
MTs have three binding sites for these compounds; however, the exact locations and drug-protein interactions of these sites are still controversial.
Structures of other complexes of 14-3-3 14-3-3 14-3-3roteins have been determined as with,37,38,39,40 but the binding sites for these proteins show less overlap with the ExoS/T-binding site than that for trehalase (Supplementary Fig. 15).
Despite advances in determining the identities of the molecules that mediate these decisions we are still incapable of predicting how simple physical parameters such as the number, position and affinity of binding sites for these molecules on the DNA determine developmental fates.
Both ERR-alpha and hepatocyte nuclear factor-4alpha are required for PGC-1alpha-mediated induction of CYP17A1, and specific binding sites for these receptors have been identified in the regulatory regions of this gene.
This conclusion is based on the fact that binding sites for these TFs were detected in families where differences between the promoter regions of SUB1A-1 (upregulated by submergence) and SUB1A-2 (non-upregulated by submergence) alleles were found.
This is surprising as the binding sites for these three miRNAs are very well conserved.
