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For much of Europe, these deep storage sites are anticipated to be sited below the sea bed on continental shelves.
Naturally, the noninhibitory NMR hits might bind elsewhere on the enzyme, but trying to anticipate this would be outside of the normal docking protocol, where a key binding site is typically targeted.
Otherwise, this binding site is regarded as an incorrectly predicted binding site.
The occurrence of the Gal binding site is apparently independent from the binding profiles of GI NoVs.
One binding site is the phosphate binding site at the catalytic cysteine residue.
The putative KNtp binding site is shown as a green circle, showing the displacement of GBM-binding sites upon nucleotide binding.
The binding site is very highly conserved.
The lower binding site was found more constructive favorable for binding.
The dexamethasone binding site was the primary binding site in the GR for all organotins.
The MEF2 binding site was identified.
Sensitivity to perturbations in ATP and gefitinib binding is anticipated because these parameters control gefitinib/ATP competition.
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