Sentence examples for binding of these complexes from inspiring English sources

In solution interactions, spectroscopic and electrochemical evidences indicate outside binding of these complexes.

All the experimental evidence suggest that the ancillary ligand 2,2′-bipyridine influences the intercalative binding of these complexes to CT DNA.

The parameters for forming the effector protein-chaperone complexes, for binding of these complexes to the TTSS, for the probability that binding leads to "injection" and for the time required for "injection" could be adjusted freely (Fig. 3A; see also supplemental online materials).

The binding of these complexes facilitates selection of cargo to be degraded, and leads to the expansion of the autophagosome and its fusion with the vacuolar membrane [ 116, 119].

The next step was to examine the binding of these complexes on effector cells, lymphocyte and monocyte populations present in PBMC as well as HL-60 cells used in ADCC assays.

Genome-wide binding and knock-down studies of Tip60-p400, NSL and MSL HAT complexes reported the binding of these complexes at Pol II positive genes and in mESCs [ 18, 19].

LPS binding to these complexes facilitates activation of the TLR4/nuclear factor (NF -κB iNF -κBatory pathway, ultinflammatorying to the pathwayion of proatherogenic cytokines inclultimatelyr necrosis factor aleadingNF-α), intorleukin-6 (IL-6), and IL-1 [ 8- 11].

Inhibitors of cyclin/Cdk complexes, by binding to these complexes, negatively regulate cell cycle progression [ 2].

DNA binding properties of these complexes were investigated and complex 1 exhibited nuclease activity.

We further explored the DNA binding ability of these complexes by an agarose gel electrophoresis study.

Isotope-edited spectroscopic analysis and binding specificity of these complexes provides information about specific enzyme transition state contacts.